In moderate communicating hydrocephalus of human fetuses, ependymal denudation is a common feature that may result in abnormal neurogenesis

doi:10.1186/1743-8454-2-S1-S5

Cerebrospinal Fluid Research

Volume 2
Suppl 1

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Jiménez AJ
Páez P
Weil B
Arráez MA
Rodríguez EM
Pérez-Fígares JM

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Páez P
Weil B
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Rodríguez EM
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This article is part of the supplement: 49th Annual Meeting of the Society for Research into Hydrocephalus and Spina Bifida .

Oral presentation

In moderate communicating hydrocephalus of human fetuses, ependymal denudation is a common feature that may result in abnormal neurogenesis

María Dolores Domínguez¹, Antonio J Jiménez¹ , Patricia Páez¹, Bernardo Weil², Miguel Angel Arráez³, Esteban M Rodríguez⁴ and José-Manuel Pérez-Fígares¹

¹Departamento de Biología Celular, Genética y Fisiología. Universidad de Málaga. E-29071 Málaga, Spain

²Servicio de Anatomía Patológica Hospital Carlos Haya, Málaga. Spain

³Servicio de Neurocirugía Hospital Carlos Haya, Málaga. Spain

⁴Instituto de Histología y Patología, Universidad Austral de Chile, Valdivia, Chile

author email corresponding author email

from 49th Annual Meeting of the Society for Research into Hydrocephalus and Spina Bifida
Barcelona, Spain. 29 June – 2 July 2005

Cerebrospinal Fluid Research 2005, 2(Suppl 1):S5doi:10.1186/1743-8454-2-S1-S5


Published:	30 December 2005

First paragraph (this article has no abstract)

Recent investigations carried out in natural and experimental mutant mice have provided strong evidence that a primary alteration of the ependymal cell lineage triggers a moderate foetal hydrocephalus [1,2]. In human cases of hydrocephalus, however, ependymal loss has been regarded as resulting from the ventricular dilatation due to the accumulation of cerebrospinal fluid [3].